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Muscle biopsies demonstrate variation in ber size, scattered necrotic and regenerating bers, and increased endomysial connective tissue Immunostaining reveals absent or diminished sarcolemmal staining with dysferlin antibodies In contrast, there may be increased cytoplasmic staining The reduced sarcolemmal immunostaining can be secondary and seen in other types of LGMD112 ; therefore, Western blot needs to be performed on the muscle or white blood cells to con rm a primary de ciency Not uncommonly, a prominent mononuclear in ammatory cell in ltrate is evident in the endomysium and surrounding blood vessels This accounts for many cases of dysferlinopathy being misdiagnosed as polymyositis113 In contrast to polymyositis, the in ammatory cells do not typically appear to invade nonnecrotic bers Another immunohistological feature that is helpful is demonstrating deposition of membrane attack complex on the sarcolemma of non-necrotic muscle bers (Fig 24 11) an early nding in dysferlinopathies and other dystrophies with in ammation that is not seen in primary in ammatory myopathies such as polymyositis, dermatomyositis, and inclusion body myositis (IBM) On electron microscopy, reduplication of the basal lamina, disruption in the sarcolemma, invaginations or papillary exophytic defects of the muscle membrane, and subsarcolemma vesicles may be appreciated114

What a convenient way to initialize an array of chars! Here s another way to accomplish the same thing:

The sarcoglycanopathies account for approximately 10% of LGMD in the following frequencies: -sarcoglycan

66%, -sarcoglycan 31%, -sarcoglycan 15%, and sarcoglycan <1%4,116,117 The clinical, laboratory, and histologic features of the sarcoglycanopathies are quite similar to the dystrophinopathies, with some children developing early onset of severe weakness resembling DMD and other patients having a later onset and slower progression similar to BMD Proximal leg and arm muscles are affected early, and calf pseudohypertrophy can often be appreciated Cardiomyopathy can also occur similar to the dystrophinopathies118

Below that line, write the following code: end_mconPress = startGame This will make the startGame function run to restart the game when you click the end_mc movie clip

char s[ 20 ] = { 'H', 'e', 'l', 'l', 'o', '\0' };

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Besides the usual dystrophic features, many muscle bers had one or more rimmed vacuoles Immunohistochemistry and Western blot analysis demonstrate a de ciency of telethonin128

LGMD 2G is caused by mutations in the telethonin gene located on chromosome 17q11 12128 Telethonin is a 19kD sarcomeric protein that is expressed in skeletal and cardiac muscles, where it localizes to the central parts of the Z-disk130 It is a ligand for the giant sarcomeric protein, titin, which helps phosphorylate the C-terminal domain of telethonin in early differentiating myocytes Telethonin may also overlap with myosin as well It is amongst the most abundant proteins in muscle The interaction of telethonin with titin appears to be important in myo brillogenesis130,131

Once again, if you leave out the dimension, the compiler will allocate just enough memory to hold your text string, including a byte to hold the zero terminator. If you include the dimension, the compiler will allocate that many array elements, and fill the array with whatever data you provide. If you provide more data than will fit in the array, your code won t compile. Here s a struct example:

Antibody testing to aid in the diagnosis of suspected in ammatory myopathies is commercially available102,125 There are varying opinions regarding its value There are typically other more speci c and expeditious means to make a diagnosis There is a frequently prolonged interval between specimen acquisition and result availability As an anecdotal impression, these tests appear to be used more frequently by rheumatologists than neurologists Although autoantibodies have been reported to occur in 60 80% of patients with idiopathic in ammatory myopathies, a select few appear to be myositis speci c and occur far less frequently102,125 Their potential value includes the distinction between potentially treatable, acquired, immune-mediated muscle disease and other myopathies including some that are hereditary in which histological evidence of in ammation may occur In addition, the presence of speci c types of antibodies

meteorInitonEnterFrame = function():Void { this_xscale += thisgrowthRate; this_yscale = this_xscale; if(this_xscale >= 150) { this_xscale -= 50; this_yscale -= 50; energy_mcplay(); energy_mcbar_mc_xscale -= 10; if(energy_mcbar_mc_xscale <= 0) { end_mc_visible = true; end_mcend_txttext = "You lose\nClick to play again"; Mouseshow(); end_mconPress = startGame; } } }

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