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not just one but lots of nine-, ten-, and eleven-year-old kids were digging in and learning to program. Cool! And the best part of all was when these kids started sending me actual shipping products that they created. You can t imagine how proud I was and still am. Over the years, I ve heard from soccer moms, hobbyists, even folks who were using the Mac for the very first time, all of whom made their way through Learn C on the Macintosh and came out the other end, proud, strong, and full of knowledge. So what do you need to know to get started Although learning C by just reading a book is possible, you ll get the most out of this book if you run each example program as you encounter it. To do this, you ll need a Mac running Mac OS X (preferably version 10.5 or later) and an Internet connection. You ll need the Internet connection to download the free tools Apple has graciously provided for anyone interested in programming the Mac and to download the programs that go along with this book. Again, if you know nothing about programming, don t worry. The first few chapters of this book will bring you up to speed. If you have some programming experience (or even a lot), you might want to skim the first few chapters, and then dig right into the C fundamentals that start in 3.

MDC ASSOCIATED WITH GENETIC DEFECTS OF STRUCTURAL PROTEINS OF THE BASAL LAMINA OR EXTRACELLULAR MATRIX

MDC 1A (ALSO KNOWN AS MDC WITH LAMININ 2 OR MEROSIN DEFICIENCY OR THE CLASSIC/OCCIDENTAL TYPE)

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Here s a quick tour of what s to come in this book: 2 takes you to Apple s web site, so you can download the free tools we ll use throughout this book. 3 starts you off with the basics of building a simple program. 4 shows you how to embed a series of programming statements into a reusable function, something you can call again and again. 5 adds variables and operators into the mix, bringing the power of mathematical expressions into your programs. 6 introduces the concept of flow control, using constructs like if, else, do, and while to control the direction your program takes. 7 covers pointers and parameters, two concepts that will add a dramatic new level of power to your programs. 8 extends the simple data types used in the first half of the book, adding the ability to work with more complex data types like arrays and text strings.

Approximately 30 40% of patients with MDC have absent or severely decreased merosin143 147 In addition, there are patients with partial merosin de ciency, analogous to the dystrophinopathies and sarcoglycanopathies145,146,148 153 However, some of these partial merosinopathies are now known to be secondary de ciencies related to glycosylation defects in -dystroglycan

all stored inside the container_mc movie clip, unloading the container_mc movie clip will automatically get rid of all the meteors At the top of the startGame function, write the code to unload the container_mc movie clip To unload a movie clip, use the unloadMovie method

LGMD 2M is caused by mutations in the POMGnT1 gene that usually causes Muscle-Eye-Brain disease, but rarely can cause a more benign form of dystrophy

9 takes this concept one step further, adding the ability to design your own custom data structures. 10 shows you how to save your program s data and read it back in again by introducing the concept of the data file. 11 covers a variety of advanced topics typecasting, unions, recursion, binary trees, and much more. Finally, 12 wraps things up and points you to the next step on your journey. Ready to get started Let s go!

Children with MDC 1A usually present at birth with generalized weakness and hypotonia birth There is a predilection for neck-, shoulder-, and hip-girdle muscles244 Calf hypertrophy may be appreciated early in the course Contractures develop, but severe arthrogryposis is rare Respiratory and feeding problems can be present but usually not severe enough to require ventilator support at birth141 Some children develop a cardiomyopathy Limited extraocular movements can be observed in the later stages Merosin-negative MDC typically has more severe weakness and is associated with a poorer prognosis compared with merosin-positive cases144 Most children with MDC 1A never ambulate independently, although rare children are able to stand and occasionally walk with assistance Individuals with only partial merosin de ciency have a milder course and can present in childhood with a DMD phenotype or in early adulthood with a phenotype similar to BMD or LGMD143 147,154 Most children with MDC 1A have normal intelligence, despite abnormal white matter changes apparent on MRI However, there is a high incidence of epilepsy (12 30%) as well as a few reported cases of occipital dysplasia in merosin-de cient CMD148,152 Epilepsy can also occur in patients with partial merosin de ciency Rare patients with MDC 1A with epilepsy and occipital agyria also have mental retardation

Serum CK levels are markedly elevated, usually over 2000 IU/L in the merosin-negative infants, while partial merosinopathies are associated with normal or mildly elevated serum CKs Brain MRI often demonstrates diffuse white matter abnormalities in T2-weighted images suggestive of dysmyelination in most children after the age of 6 months (Fig 24 13) Additionally, occipital polymicrogyria/agyria and hypoplasia of pons and/or cerebellum are evident in rare cases141 Patients with partial merosin de ciency may or may not have cerebral hypomyelination on MRI Visual- and somatosensoryevoked potential may reveal delayed latencies in MDC 1A155 Slowing of nerve conduction velocities is also commonly appreciated147

function startGame():Void { container_mcunloadMovie(); Mousehide(); cursor_mcstartDrag(true); cursor_mc_visible = true; meteorInterval = setInterval(_root, "createMeteors", 1000); end_mc_visible = false; }

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Figure 24 13 Congenital muscular dystrophy T2-weighted MRI of brain of an infant with merosin-negative congenital muscular dystrophy reveals increased signal of the subcortical white matter consistent with hypomyelination

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