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As you can see, the first line checks to see if the opt1 field is empty. If it is not, we construct a link element that passes a variable called action to the deck results.asp with the value of the field opt1. If the field is empty, we write nothing at all. In this way, we can build a menu that contains only the user s options, with no dummy statements or spaces on the display. There are five of these IF statements, or one for each option. If we had designed the database differently for example, with a table that links to the user ID and allows any number of options instead of restricting it to five we could have just built the menu with a simple loop through the second table, but we would also have had to create a second Recordset. In the interests of simplicity I have done it this way. We will see a more complex type of interaction later, in results.asp. If we run the application with what we have done so far, you will see that we have a menu appear with just two items on it Preferences and Home. So far, so good. Now let s allow the users to add their preferences.

Web Item Parameters URL Parameterization Template Settings for Web Item Parameters Reusable Web Item Parameters

Question 11-5

F i g . 4 7-3

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hyperre exic state, such as the Hoffmann sign and the crossed adductor re ex of the thigh muscles Also, re exes may be inverted, as in the case of a lesion of the fth or sixth cervical segment; here the biceps and brachioradialis re exes are abolished and only the triceps and nger exors, whose re ex arcs are intact, respond to a tap over the distal radius In addition to hyperactive phasic myotatic re exes ( tendon jerks ), certain lesions, particularly of the cervical segments of the spinal cord, may result in great enhancement of tonic myotatic re exes These are stretch re exes in which a stimulus produces a prolonged asynchronous discharge of motor neurons, causing sustained muscle contraction As the patient stands or attempts voluntary movement, the entire limb may become involved in intense muscular spasm, sometimes lasting for several minutes.

Code 102 152 202 222 472 502 562 682 103 203 223 333 473 503 563 683 104 204 224 334 474 564 105 205

During this period the limb is quite useless Presumably there is both an interruption of lateral reticulospinal inhibitory in uences on the anterior horn cells and a release of the medial reticulospinal facilitatory effects needed in antigravity support (Henneman) With bilateral cerebral lesions, exaggerated stretch re exes can be elicited in cranial as well as limb and trunk muscles because of interruption of the corticobulbar pathways These are seen as easily triggered masseter contractions in response to a brisk downward tap on the chin (jaw jerk) and brisk contractions of the orbicularis oris muscles in response to tapping the philtrum or corners of the mouth In advanced cases, weakness or paralysis of voluntary movements of the face, tongue, larynx, and pharynx are added (bulbar spasticity or pseudobulbar palsy; see pages 426 and 445).

function: Solution. We can first express ) as an anonymous ./('T > > f = G ( x ) a . 2 + 2 5 * x - 2 0 0 * x . " 2 + 6 15 * x . ^ 3 9 0 0 * x . ^ 4 + 4 0 0 * x . ^ 5 ; valuesof the independent and dependent a We can then generate seriesof equally spaced variables: >> x=0:0.1 :0.8;

where b = Rb a Ra a b is the representation of the matrix with respect to frame b. (2.23)

There have been many investigations of the biochemical changes that underlie spasticity and the mechanisms of action of antispasticity drugs These have been reviewed by Davidoff Since glutamic acid is the neurotransmitter of the corticospinal tracts, one would expect its action on inhibitory interneurons to be lost As mentioned earlier, gamma-aminobutyric acid (GABA) and glycine are the major inhibitory transmitters in the spinal cord; GABA functions as a presynaptic inhibitor, suppressing sensory signals from muscle and cutaneous receptors Baclofen, a derivative of GABA, as well as diazepam and progabide, are thought to act by reducing the release of excitatory transmitter from the presynaptic terminals of primary afferent terminals Actually, none of these agents is entirely satisfactory in the treatment of spasticity when administered orally; the administration of baclofen intrathecally may have a more bene cial effect.

FIGURE 11-2

Glycine is the transmitter released by inhibitory interneurons and is measurably reduced in quantity, uptake, and turnover in the spastic animal There is some evidence that the oral administration of glycine reduces experimentally induced spasticity, but its value in patients is uncertain Interruption of descending noradrenergic, dopaminergic, and serotonergic bers is undoubtedly involved in the genesis of spasticity, although the exact mode of action of these neurotransmitters on the various components of spinal re ex arcs remains to be de ned Table 3-1 summarizes the main attributes of upper motor neuron lesions and contrasts them with those of the lower motor neuron..

FIGURE 2-5

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